Sunday, June 2, 2013

NEUROTOXIC DISORDERS

The infections in my body seem to be stable as long as I'm on the antivirals (high dose) and antibiotics (low dose). 

Right now my main issues are primarily neurological and toxin induced so I'll probably be focused on toxins and detoxing for a bit. 

There's some good info in this blog post written by Wayne Anderson ND (he's with Gordon Medical and Associates). 

Interesting that they are starting to look at CD57 count, typically used by LLMD's for assessing an aspect lyme disease. They are realizing the CD57 count can be used as a an indicator of mold illness.

It's worth reading the whole post and a great blog to sign up to get treatment information and updates:

Certain types of toxigenic mold can also be a major factor in neurotoxic conditions. Petrol-based pesticides, insecticides, and solvents are a third major contributor to neurotoxic inflammation. The extensive neurotoxic effects of pesticides, for example, have been well documented. Heavy metals are sludge toxins with some of the same neurotoxic effects as petrol-based chemicals. Mercury, lead, cadmium, and arsenic involve similar neurotoxic mechanisms of action and can cause similar symptoms.
Many of these intracellular pathogens proliferate very slowly, over months, years, or even decades. They gain access to the body’s cells through a simple process and can remain for years.
Step 1. Toxic build-up: Following exposure, the toxin is absorbed through the cell membrane and penetrates the cell. Our own cell membranes consist of two layers of fat (bilipid), which normally provide very effective gatekeeper functions, allowing into the cell what should be allowed as a nutrient and repelling what should be rejected as a toxin. However, because the pathogens also contain lipid molecules, they confuse the gatekeeper mechanism of cell membranes, penetrating the cell wall, and essentially hijacking the cell. 
Step 2. Immune reactions: The immune system is activated and tries to destroy or remove the toxins harbored within the cell, but frequently cannot do so. When the system cannot break up or destroy these microbes, inflammation results. As these efforts by the immune system become more and more intense, activity shifts from cellular immunity to antibody production. This frantic immune activity is often associated with the symptoms of autoimmune conditions. 
Step 3. Attempts at detoxification: Neurotoxins are typically detoxified through the methylation pathway. This pathway involves numerous genetic strands that all coordinate to remove waste from within the cell, through the cell membrane, to move toxins back into the bloodstream. 
If toxins build up within the cell, the cell’s function is diminished. These irritants can dysregulate the mitochondria, lowering energy production by the cell. No matter where the cell is located in the body, there will be reduced function or even lack of function. If the toxic cells are stored in muscle tissue, that may be experienced as weakness, pain, or an array of symptoms resembling fibromyalgia. If the toxins have accumulated within the brain, that will create symptoms related to functional neurological deficits such as impaired memory, cognitive processing, and brain fog, as well as mood instability resulting in depression or anxiety.

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